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肝细胞损害时呈现“循环机能亢进”状态,肝硬变时心排出量达到正常人的2—4倍。在急性肝炎时随着肝细胞的恢复,,这种循环机能亢进状态也正常化。肝硬化时高输出量的机制与小动脉的扩张、末稍动静脉侧支循环的开放有关,如肝掌、蜘蛛痣等也是其中的表现。另外因为末稍血管弹性有降低的趋势,肝脏疾病患者的血压正常或者有低下倾向。
Liver cell damage presents “hyperfunction” state, liver cirrhosis, cardiac output 2-4 times normal. With the recovery of hepatocytes in acute hepatitis, this state of hyperfunction of circulation is also normalized. The mechanism of high output in cirrhosis is related to the expansion of arterioles and the opening of distal arteriovenous collateral circulation, such as liver palms and spider moles. In addition, due to the decrease of distal vascular elasticity, blood pressure in patients with liver diseases is normal or has a tendency to be depressed.