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甜菜细胞质的雄性不育性(CMS)是受制于一种细胞质因素和至少两个核基因的相互作用。有迹象表明,引起细胞质雄性不育反应的可能是某些类似病毒的粒子。通过热处理(36℃以上)和随后分生组织培养可能从细胞质雄性不育植株中得到雄性能育植株。多数支持这个假设,即这种新的雄性能育性取决于含 RNA 的粒子的疏化或清除。今后的研究需要澄清是否从一切 CMS——植株都能产生一种雄性能育的对应植株,而且是否这些新的能育植株都适合于做 O 型系。OWen(1942)首先描述了甜菜细胞质雄性不育(CMS)的遣传,他认为这种雄性不育性是受制于一种细胞因素和至少两个或者可能更多的孟德尔式因子。凡带有所谓“雄性不育”或(S)一原生质和隐性恢复基因的植物都是雄性不育的。正常的原生质(N)以及或者显性恢复基因导致雄性能育性。一株具有(N)原生质和隐性恢复基因的植物可以用来授粉以建立一个雄性不育系,这样的授粉株便是通常所称的“O”型系、零恢复系或保持系,因为它除保持不育性外不恢复雄性能育性,
Cytoplasmic male sterility (CMS) in sugar beets is governed by a cytoplasmic factor and the interaction of at least two nuclear genes. There are indications that some of the virus-like particles may cause cytoplasmic male sterility reactions. Male fertile plants may be obtained from cytoplasmic male sterile plants by heat treatment (above 36 ° C) and subsequent meristem culture. Most support the hypothesis that this new male fertility depends on the scavenging or clearance of RNA-containing particles. Future studies will need to clarify whether a male-fertile counterpart can be produced from all CMS-plants and whether these new fertile plants are suitable for the O-line. OWen (1942) first described the removal of the cytoplasmic male sterility (CMS) of sugar beet, which he believes is limited by one cytokine and at least two or possibly more Mendelian factors. Plants with so-called “male sterility” or (S) a protoplast and a recessive restorer gene are all male-sterile. Normal protoplasts (N) and / or dominant -recovering genes lead to male fertility. A plant with a (N) protoplast and a recessive restorer gene can be used for pollination to create a male sterile line, which is what is commonly referred to as the “O” type, zero restorer, or maintainer because It does not restore male fertility except for sterility,