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多形核白细胞(PMN)是宿主对感染的早期反应中主要的细胞效应器,其半寿期为5～6h,随即经凋亡过程而死亡。在炎症反应处,PMN在细胞氧化还原状态改变后发生一些细胞变化。已知巯基抗氧化剂还原性谷胱甘肽维持氧化还原潜能和保护细胞免受氧化损害。激
Polymorphonuclear leukocytes (PMNs) are the major cellular effectors of early host response to infection with a half-life of 5-6 h, which is followed by an apoptotic process. At the inflammatory response, PMN undergoes some cellular changes after the cellular redox status changes. It is known that thiol-resistant reductant glutathione maintains the redox potential and protects cells from oxidative damage. Excited