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患者男,50岁,医生。因心前区持续疼痛3天,由当地医院转入湖南医科大学附二医院。既往有高血压病史和大量吸烟史。入院体查:BP 150/110mmHg,急性痛苦病容,查体欠合作,心脏向左下扩大,HR48次/分,律不齐,余(-)。当地心肌酶学示:AST126.2 U/L,LDH274.6 U/L,α-HBDl80.9 U/L,CK473.7 U/L,CK-MB333.2 U/L;入院急查心肌酶学示 AST805U/L,LDH1337.2 U/L,α-HBD230.4 U/L,CK53.4U/L,CK-MB11.6 U/L。ECG 呈动态改变,支持“下壁心肌梗塞并Ⅲ°AVR”诊断。经扩冠抗凝,酌情降压及支持对症治疗,患者胸痛症状消失,心电图明显改善,
Patient male, 50 years old, doctor. Because of precordial continuous pain for 3 days, transferred from the local hospital attached to the Second Affiliated Hospital of Hunan Medical University. Previous history of hypertension and a large number of smoking history. Admission physical examination: BP 150 / 110mmHg, acute pain, physical examination owed cooperation, the heart expanded to the lower left, HR48 beats / min, irregular laws, Yu (-). Local myocardial enzymes showed: AST126.2 U / L, LDH274.6 U / L, α-HBD80.9 U / L, CK473.7 U / L, CK-MB333.2 U / L; The study showed AST805U / L, LDH1337.2U / L, α-HBD230.4U / L, CK53.4U / L, CK-MB11.6U / L. ECG was dynamically changed to support “lower myocardial infarction and Ⅲ ° AVR” diagnosis. After the crown anticoagulant expansion, as appropriate, antihypertensive and support symptomatic treatment, patients with chest pain disappeared, ECG was significantly improved,