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五羟色胺较重要的来源是小肠嗜铬细胞,释放至大循环经血小板运输后很快被肺和肝床清除.目前已知有四种五羟色胺受体中亚型赋有特殊的生理应签,可使血小板聚合,引起外周血管收缩增强其他神经体液的血管收缩调节作用,曾有报导先兆子痫妇女血小板和五羟色胺浓度减低.投与五羟色胺可导致血管痉挛、血压高、尿少.本文作者证实ketanserin可控制先兆子痫的高血压,所选资料归定的标准包括高血压、尿蛋白、血小板的减少和中枢神经系统症状.如妊娠前有高血压史或妊娠早、中期的早期,平均动脉压90～95mmHg为慢性高血压合并先兆子痫,如无高血压史则为单纯的先兆子痫.
The most important source of serotonin is small intestine chromaffin cells, release to the large circulation platelet transport by the lungs and liver bed clearance soon.It is known that there are four serotonin receptor subtypes endowed with a special physiological should sign, so that platelets Polymerization, causing peripheral vasoconstriction enhances the vasoconstriction regulation of other neurohumoral effects, there have been reports of preeclampsia in women platelets and serotonin concentrations decreased.Citrate administration of serotonin can lead to vasospasm, high blood pressure, oliguria.The authors confirm that ketanserin can control pre-eclampsia Epilepsy, hypertension, hypertension, urinary protein, thrombocytopenia and central nervous system symptoms, such as a history of hypertension before pregnancy or early, middle early pregnancy, mean arterial pressure of 90 ~ 95mmHg Chronic hypertension with preeclampsia, if no history of hypertension is a simple pre-eclampsia.