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目的 研究创伤性急性肺损伤 (AL I)家兔体内肿瘤坏死因子 (TNF-α)的变化 ,探讨山莨菪碱 (6 5 4- 2 )对创伤性 AL I及多器官功能障碍 (MODS)的防治作用及可能机制 .方法 采用家兔创伤性 AL I模型 ,实验动物随机分为正常对照组 (8只 )、创伤模型组 (10只 )、 6 5 4- 2治疗组 (
Objective To investigate the changes of tumor necrosis factor-α (TNF-α) in rabbits with traumatic acute lung injury (ALI) and to explore the effect of anisodamine (6 5 4- 2) on traumatic AL I and multiple organ dysfunction Prevention and treatment and possible mechanism.Methods Rabbit traumatic ALI model was divided into normal control group (n = 8), traumatic model group (n = 10) and treatment group