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谷氨酸δ2受体(GluRδ2)主要在浦肯野细胞中表达,对小脑功能有决定性作用。GluRδ2基因缺陷的小鼠表现共济失调和运动学习功能损伤,而且,还表现平行纤维与浦肯野细胞接合处的长时程抑制消失及浦肯野细胞中平行纤维(PF)
The glutamate δ2 receptor (GluRδ2) is mainly expressed in Purkinje cells and plays a decisive role in cerebellar function. GluRδ2-deficient mice show ataxia and motor function impairment, and also show the long-term inhibition of disappearance of parallel fibers to Purkinje cells and parallel fibers (PF) in Purkinje cells