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目的研究慢性输卵管炎易导致输卵管妊娠的生化机制。方法应用ABC免疫组化技术,以6种生物素化的凝集素为探针,对慢性输卵管炎粘膜糖食复合物进行了标记,并与正常及妊娠输卵管粘膜糖复合物进行了比较。结果发现慢性炎之输卵管较正常输卵管粘膜糖食。复合物的表达有明显不同,而与妊娠输卵管粘膜糖复合物的表达接近或相同。结论慢性输卵管炎时粘膜糖复合物的改变是其易致输卵管妊娠的因素之一。
Objective To study the biochemical mechanism of tubal pregnancy induced by chronic salpingitis. Methods ABC immunohistochemical technique was used to probe into the chronic salpingitis mucosal confectionery with 6 kinds of biotinylated lectins as probe, and compared with normal and pregnancy tubal mucosal glycoconjugates. The results found that tubal chronic tuberculosis mucosal sugar than normal. The expression of the complex significantly different, but with the pregnancy tubal mucosal glycoprotein expression close or the same. Conclusion The change of mucosal saccharide complex in chronic salpingitis is one of the factors that lead to tubal pregnancy.