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维生素D可能通过抑制心肌肾素血管紧张素系统(renin-angiotensin system,RAS)和成纤维细胞生长因子(fibroblast growth factors,FGFs)改善尿毒症所致的心肌肥厚。在实验性大鼠尿毒症模型中,研究者比较利用维生素D类似物帕立骨化醇和血管紧张素转换酶抑制剂依那普利抑制RAS和FGF受体基因表达和心肌肥厚改善之间的关系。方法:给予切除5/6
Vitamin D may improve uremia-induced myocardial hypertrophy by inhibiting renin-angiotensin system (RAS) and fibroblast growth factors (FGFs). In an experimental rat uremic model, investigators compared the use of the vitamin D analog paricalcitol and the enoxaparin inhibitor of inhibition of RAS and FGF receptor gene expression and improvement of myocardial hypertrophy . Method: Give resection 5/6