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老年聋呈进行性的高频听力下降,且缓慢地向低频扩展。Zwardemaker氏在1899年首先描写了老年聋的发展规律。1964～1965年先后有学者报告老年聋患者耳蜗核细胞数目比正常人减少,听通路核团内细胞有退变现象,推论这种神经细胞的衰老改变是老年聋发生的主要因素。本研究是在高倍显微镜下分别计算耳蜗背核和腹核的神经细胞数目,探讨老年聋患者的耳蜗核是否有损失。所用脑组织标本是取自老人医院、经耳科检查及纯音测听、临床随诊排除了其他致聋病因而确诊为老年聋后因心脏病或肺炎死亡的6例尸体,其年龄在76～89岁之间,平均听力损失22至77dB不等。尸体放置在4℃的冷藏室,
Senile deafness showed progressive high-frequency hearing loss, and slowly extended to low-frequency. Zwardemaker first described the development of deafness in 1899. Some scholars from 1964 to 1965 reported that the number of cochlear nuclei in deaf patients was lower than that in normal subjects, and degeneration of cells in the auditory nucleus was observed. It is concluded that the change of senescence of these neurons is the main factor of senile deafness. In this study, the number of nerve cells in the dorsal and ventral nucleus of the dorsal horn was calculated under a high magnification microscope to investigate whether there is any loss of the cochlear nucleus in elderly deaf patients. The brain tissue samples taken from the elderly hospital, the ear examination and pure tone audiometry, clinical follow-up rule out other causes of deafness and thus diagnosed as senile deafness due to heart disease or pneumonia after the death of 6 cases of the body, aged 76 ~ Between 89 years of age, the average hearing loss ranged from 22 to 77 dB. The body was placed in a freezer at 4 ° C,