听力突然丧失伴有耳鸣、眩晕者是耳科诊断中的一个难题,大部分病例的病因不明,部分病例体力劳动和周围环境压力的变化是促发原因。真正的组织病理学变化尚未明瞭,但动物实验提示脑脊液和外淋巴液之间的压力相互有关,当蜗窗受损时导致内淋巴管压力增高、耳蜗破坏和内耳病理生理变化。本文介绍二种内耳损伤的机制一、胸或腹腔压力增高或头部静脉充盈使脑脊液压力增高,这种压力通过耳蜗导水管或Ⅶ、Ⅷ颅神经的神经鞘传至外淋巴间隙,因此膜迷路就像蜗窗和镫骨底板一样暴露于突然增高的压力 A sudden loss of hearing associated with tinnitus and vertigo is a difficult problem in the diagnosis of otology. In most cases, the etiology is unknown. In some cases, manual labor and changes in ambient pressure are the contributing factors. True histopathological changes are not yet clear, but animal experiments suggest that the pressure between cerebrospinal fluid and perilymph is related to each other, leading to increased pressure on the lymphatic vessels, destruction of the cochlea and pathophysiological changes of the inner ear when the snails are impaired. This article describes two mechanisms of damage to the inner ear, thoracic or abdominal pressure increases or filling the head of the cerebrospinal fluid pressure increases, this pressure through the cochlear aqueduct or Ⅶ, Ⅷ cranial nerve sheath to the lymphatic space, so the labyrinth Just as the snail and stapes are exposed to sudden, elevated pressures