有许多因素指出煤矿工人的尘肺、矽肺和其它慢性肺部疾患均有免疫反应参与。在这些疾病中,当刺激因子与宿主相互作用时期,免疫系统以不同方式参与了作用。例如,实验性结核能产生与宿主肺组织成分起反应的抗体。给患结核动物以抗肺血清,改变感染过程,这种动物表现了一种对结核菌抵抗力降低的倾向,并更多地发展为粟粒性结核,证明抗体参与肺结核的发生。也曾对其它慢性肺部疾病,包括实验性矽肺,进行这样的观察。本文目的是以各方面证据来说明在尘肺的疾病 There are many factors that indicate coal miners’ pneumoconiosis, silicosis and other chronic lung diseases are involved in immune responses. In these diseases, the immune system participates in different ways during the period when the stimulus interacts with the host. For example, experimental tuberculosis can produce antibodies that react with the components of the host’s lung tissue. To give TB animals anti-pulmonary sera and to change the course of the infection, this animal showed a reduced resistance to M. tuberculosis and more to miliary TB, demonstrating that the antibodies are involved in tuberculosis. This observation has also been made on other chronic lung diseases, including experimental silicosis. The purpose of this article is to show evidence of pneumoconiosis in all its aspects