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目的：探讨二硫化碳对周围神经损伤的发展规律。方法：对长期接触二硫化碳浓度高于国家最高容许浓度１０ｍｇ／ｍ３数倍的５７例工人，１９８９、１９９５年进行临床和神经肌电图检查。结果：６年后，在５７例工人中１８例肯定周围神经病转为可疑或恢复正常。在１２例已脱离接触二硫化碳半年至１７年的工人中，８例仍为肯定周围神经病，２例转为可疑，１例恢复正常。结论：二硫化碳慢性中毒性周围神经病恢复是缓慢的，反映了二硫化碳所致的轴索损伤修复慢的特征。我们主张神经肌电图检查一旦发现周围神经病，应及时脱离以利于神经损伤的恢复
Objective: To investigate the development of peripheral nerve injury caused by carbon disulfide. Methods: 57 workers whose long-term exposure to carbon disulfide concentrations were several times higher than the national maximum allowable concentration of 10 mg / m3 were examined by clinical and neuro-EMG in 1989 and 1995. RESULTS: Six years later, 18 of 57 workers confirmed that peripheral neuropathy turned suspicious or returned to normal. Among the 12 workers who had been out of contact with carbon disulfide for six months to 17 years, 8 remained positive peripheral neuropathy, 2 became suspicious and 1 returned to normal. CONCLUSIONS: Chronic carbon disulfide chronic toxic peripheral neuropathy recovery is slow, reflecting the slow recovery of axonal injury due to carbon disulfide. We advocate neuromuscular examination once found peripheral neuropathy, should be promptly removed in order to facilitate the recovery of nerve injury