论文部分内容阅读
长期血透病人基础心排出量升高。这一高动力循环状态的持续将引起心室舒张期容量增加和心肌收缩功能不良。高动力状态可能是引起血透病人充血性尿毒症性心肌病的决定因素。体液失衡、血液动静脉分流、贫血是增加心排出量的主要因素。使用重组人类促红细胞生成素(fhEPO)治疗,可缓慢纠正继发于慢性肾衰的贫血,这使研究贫血在高动力状态中所起作用成为可能。由此,作者研究12例血透病人用 rhEPO 治疗最初四个月的心排出量的演变。
Long-term hemodialysis patients with increased basal cardiac output. The persistence of this hyperdynamic circulation will lead to increased ventricular diastolic volume and poor myocardial contractility. High dynamic state may be the cause of hemodialysis patients with congestive uremic cardiomyopathy, a determinant. Body fluid imbalance, arterial shunt, anemia is the main factor to increase cardiac output. Treatment with recombinant human erythropoietin (fhEPO) can slowly correct anemia secondary to chronic renal failure, making it possible to study the role of anemia in high-powered states. As a result, the authors studied the evolution of cardiac output in the first four months of 12 hemodialysis patients treated with rhEPO.