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为了探讨稻白叶枯细菌不同毒力株诱导抗病性的差异及其与寄主互作过程中的活性氧代谢的变化，选用抗病品种湘早籼３号（湘３）和感病品种浙辐８０２（浙８））为材料，在人工气候条件下培养至６叶期，分别用稻白叶枯细菌的弱毒株（７５－１）和强毒株（７６－２５）在第５叶剪叶接种诱导后，１２，２４，４８，７２ｈ测定诱导叶中ＳＯＤ活性、ＭＤＡ含量和Ｏ－·２产生速率的动态变化；并于诱导后第６ｄ在第６叶挑战接种，１５ｄ后调查诱导效果．结果表明，２种菌株均使２个水稻品种产生了系统获得性抗性（ＳＡＲ），７５－１对湘３和浙８的诱导及７６－２５对湘３的诱导均类似于非亲和性相互作用，即诱导叶中ＳＯＤ活性降低，Ｏ－·２产生速率及ＭＤＡ含量升高．Ｏ－·２产生速率于诱导后１２ｈ增加到较高水平，ＭＤＡ含量逐渐增高，ＳＯＤ活性自１２ｈ开始下降，４８ｈ前后降至最低点．７６－２５对浙８的诱导与上述情况不同，诱导叶中ＳＯＤ活性平均升高９．１％，Ｏ－·２产生速率平均降低５．８％，这种变化类似于亲和性相互作用．结果提示，不同毒力的菌株均可启动抗病防卫反应系统基因，但启动机制可能有差异．
In order to investigate the difference of resistance induced by different virulent strains of rice leaf blight and the change of active oxygen metabolism during the interaction with host, the selection of resistant varieties Xiangsanxian 3 (Xiang 3) and susceptible varieties Zhejiang Spike 802 (Zhe 8) was cultured under the artificial climatic conditions until the 6-leaf stage. The 5-leaf scissors (75-1) and virulent strains (76-25) After induced by leaf inoculation, the dynamic changes of SOD activity, MDA content and O-2 production rate in leaves were detected at 12, 24, 48 and 72 h after induction, challenge challenged on the 6th leaf on the 6th day after induction, and the induction effect was investigated 15 days later . The results showed that both of the two strains produced systemic acquired resistance (SAR) in two rice cultivars, the induction of 75-1 to Xiang 3 and Zhe 8 and the induction of 76-25 to Xiang 3 were similar to those of non-affinity Interaction, that is induced leaves SOD activity decreased, O- • 2 production rate and MDA content increased. The production rate of O · 2 increased to a higher level at 12h after induction, and the content of MDA gradually increased. The activity of SOD decreased from 12h to the lowest after 48h. The induction of Zhejiang Zhe 8 by 76-25 was different from that of the above. The SOD activity in the induced leaves increased by 9.1% and the rate of O · 2 production decreased by 5.8% on average, which was similar to the affinity interaction. The results suggest that strains with different virulence may activate the genes of the defense response system, but the mechanism may be different.