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患者男,70岁,1991年11月9日因冠心病,完全性房室传导阻滞,反复发作尖端扭转型室性心动过速伴阿斯发作安置进口VVI起搏器。电极送达右心室尖部测初始起搏阈值:脉宽0.5ms,感知2mV,输出≥1mA见起搏电信号全部带动QRS-T波,感知功能正常。术后1月,病人因再发昏倒入院,心电图示间歇性心室起搏信号脱失及感知不足。X线检查未见明确电极移位。12月11日打开囊袋,见脉冲发生器复盖螺旋之硅胶帽盖脱落。起搏导线头与连接器塑料套内充满血液。用导引钢丝插入导线内排出导线内腔血液,擦干导线尾端及连接管腔血液。测电极心室起搏阈值:脉宽0.5ms,感知2mV,输出≥2mA,见起搏、感知功能正常。将导线连于起搏器上,恢复正常起搏感知,随访至今。
Male patient, 70 years old, November 9, 1991 due to coronary heart disease, complete atrioventricular block, recurrent torsades de pointes ventricular tachycardia with Aspen placement of imported VVI pacemaker. The electrode reaches the tip of the right ventricle measured initial pacing threshold: 0.5ms pulse width, sensing 2mV, the output ≥ 1mA see all pacing electrical signals to drive QRS-T wave, the sensing function is normal. One month after the operation, the patient was admitted to hospital due to recurrent coma, ECG showed intermittent ventricular pacing signal loss and lack of awareness. X-ray examination showed no clear electrode displacement. Open the pouch on December 11 and see the silicone cover of the pulse generator cover screw off. Pacing leads and connector plastic sleeve filled with blood. Use the guide wire to insert the guide wire into the inner lumen of the guide wire, wipe the end of the guide wire and connect the lumen blood. Electrode ventricular pacing threshold: 0.5ms pulse width, sensing 2mV, the output ≥ 2mA, see pacing, sensing function is normal. The wire connected to the pacemaker, resume normal sense of pacing, follow-up so far.