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实验研究已证实免疫炎症反应参与了急性冠脉综合征发生发展的病理过程,CD40-CD40L系统作为免疫炎征反应中细胞信息通道的关键介导因子,在急性冠脉综合征发生、发展病理过程中发挥了重要作用。深入研究CD40—CD40L系统在急性冠脉综合征发生发展中的作用机制,对急性冠脉综合征的预防及寻找新的治疗靶点有重要理论及临床意义。
Experimental studies have confirmed that immune-inflammatory responses are involved in the pathogenesis of the development of acute coronary syndromes. The CD40-CD40L system acts as a key mediator of cellular signaling pathways in the immune response to inflammation and develops in pathological processes of acute coronary syndrome Has played an important role. In-depth study of CD40-CD40L system in the occurrence and development of acute coronary syndrome mechanism, the prevention of acute coronary syndrome and find new therapeutic targets have important theoretical and clinical significance.