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消化性溃疡主要指胃和十二指肠的慢性溃疡性病变,在人类的远古历史上就有记载。随着近年来消化道生理学的进展,以及临床上内窥镜技术以及各种新型制酸剂的使用,对于消化性溃疡的认识已逐步深入。50年代中期,Hollander就以胃自身何以不被胃液所消化为命题,概括了直接决定消化性溃疡能否产生的两个方面:一是胃液中盐酸和胃蛋白酶有腐蚀消化道粘膜产生溃疡的作用,二是正
Peptic ulcer mainly refers to chronic ulcerative lesions of the stomach and duodenum, recorded in the ancient history of mankind. With the progress of gastrointestinal physiology in recent years, as well as clinical endoscopic technology and the use of various new antacids, the understanding of peptic ulcer has been gradually deepened. In the mid-1950s, Hollander gave a proposition as to why the stomach itself is not digested by gastric juice. It summarizes two aspects that directly determine whether peptic ulcer can be produced. First, hydrochloric acid and pepsin in the gastric juice corrode the digestive mucosa to produce ulcer Second, positive