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妊娠中毒症的胎儿预后不良,作者报告围产期死亡为10.4%,比非妊毒症组的2.1%高5倍。其中除胎盘早剥外多为未熟儿、小于孕令的胎儿(SFD),其发病率为26.3%,重症妊毒症的未熟儿,SFD的发病率尤高占56.5%。未熟儿发生的主要原因是子宫胎盘循环障碍:基于妊毒症的主要病理生理学改变为小动脉痉挛,在子宫胎盘可发生血流障碍,局部呈缺血状态,导致胎盘功能低下,母儿间物质代谢不良,胎儿发育障碍。鹤岗做动物实验性高血压,作者作动物实验性食盐负荷试验,皆可引起胎儿发育迟缓,因此认为子宫内生长迟缓儿(IUGR)与子宫胎盘血流量降低有密切关
The fetus with gestational toxicosis has a poor prognosis. The authors report that perinatal mortality is 10.4%, which is 5 times higher than that of non-PIH patients. Except for placental abruption, most of them were infantile infants, less than gestational age fetus (SFD), the incidence was 26.3%. Infantile immature infants with severe PIH had the highest incidence of SFD (56.5%). Unripe infants mainly due to uterine placenta circulatory disorders: based on the main pathophysiology of pregnancy-induced changes in arteriolar spasm, uterine placenta can occur in blood flow disorders, local ischemia, resulting in poor placental function, maternal and child substances Metabolic disorders, fetal developmental disorders. Hegang animals for experimental hypertension, the author of animal experimental salt load test, can cause fetal retardation, so that intrauterine growth retardation (IUGR) and uterine placental blood flow decreased closely related