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内源性神经介质系统在缺氧缺血性脑损伤的病理生理中起着重要的作用。腺苷 (ＡＤＯ)则是其中一种作用较强的神经调节因子[1] 。为了探讨腺苷在新生儿缺氧缺血性脑病(ＨＩＥ)中的作用及其作用机制 ,本研究利用 7日龄ＳＤ大鼠5 4只 ,制备了新生大鼠缺氧缺血性脑损伤的动物模型 ,
Endogenous neurotransmitter system plays an important role in the pathophysiology of hypoxic-ischemic brain damage. Adenosine (ADO) is one of the more potent neuromodulators [1]. In order to explore the role of adenosine in neonatal hypoxic-ischemic encephalopathy (HIE) and its mechanism of action, we used 5 4-day-old SD rats to prepare neonatal rats with hypoxic-ischemic brain damage Animal model,