休克时可出现急性呼吸衰竭,并往往有肺脏的病理形态学改变。本文观察了两种不同实验模型肺的形态学变化,并对变化初步加以探讨。 健康家兔22只,分为两组:甲组(SMAO休克组)造成小肠缺血一小时,松夹后恢复血流导致肠系动脉膜上动脉闭塞性休克。乙组(RDS组)自家兔耳静脉注入油酸(0.08ml/kg)复制RDS。待实验例死亡后,肉眼观察小肠及肺脏。每例肺标本均取材,H.E.染色,普通光镜下观察。 Acute respiratory failure can occur during shock and often have pathological changes in the lungs. In this paper, morphological changes of lungs were observed in two different experimental models, and the changes were initially discussed. Twenty-two healthy rabbits were divided into two groups: Group A (SMAO shock group) caused intestinal ischemia for one hour, and then the blood flow was restored after the loose clip to cause the occlusive shock on the superior mesenteric artery. Group B (RDS group) rabbits ear vein injection of oleic acid (0.08ml / kg) copy RDS. After the death of the experimental case, the small intestine and lungs were visually observed. Every case of lung specimens were taken, H.E. staining, under ordinary light microscope.