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老年性白内障的发病机理与许多因素有关,但年龄增加,晶体赤道部构造损坏,是最重要的因素。Wistar大鼠增龄性白内障可作为老年性白内障的模型。长期给与抗白内障药物,不影响晶体上皮细胞的活性。在添加硒的培养液中培养的晶体上皮细胞的超氧化物岐化酶(SOD)活性下降。此外,添加硒的培养液中再加GSH或Pirenoxin培养时,SOD活性处于正常范围。另外,观察了Wistar-京都系雄性自发性高血压(SHR)大鼠晶体混浊,并将其混浊度分为五级。
The pathogenesis of senile cataract and many factors related to age, but the crystal structure of the equator is damaged, is the most important factor. Age-related cataract in Wistar rats can be used as a model of senile cataract. Long-term anti-cataract drugs, does not affect the activity of crystalline epithelial cells. Crystalline epithelial cells cultured in selenium-added culture medium have decreased superoxide dismutase (SOD) activity. In addition, the addition of selenium in culture medium plus GSH or Pirenoxin culture, SOD activity in the normal range. In addition, the crystal turbidity of the Wistar-Kyoto male spontaneous hypertension (SHR) rats was observed and its turbidity was divided into five levels.