尿崩症可为原发性,或由于转移性脑肿瘤、白血病、肉芽肿性疾病、感染和外伤引起神经垂体系统损害。本文报告2例在心跳骤停后发生尿崩症。 例1,女,32岁,因甲亢伴甲状腺危象、肺炎入院,入院后几小时发生室性心动过速,继之心跳呼吸骤停,经电复律恢复窦性心律,但仍无反应,需用机械通气。以后5天中,尿量进行性增至7升/天。第8天血清钠升至169毫当量/升,血清渗透性为339毫渗量/升,尿比重1.005,诊断为尿崩症。第9天开始用后叶加压素15单位肌注,尿量从300毫升/小时降至85毫升/小时。以后4天仍间歇肌注维持。第13天开始改用静脉输 Diabetes insipidus may be primary, or cause damage to the neurohypophysis due to metastatic brain tumors, leukemias, granulomatous diseases, infections and trauma. This article reports two cases of diabetes insipidus after a sudden cardiac arrest. Case 1, female, 32 years old, due to hyperthyroidism with thyroid crisis, admission of pneumonia, a few hours after admission, ventricular tachycardia occurred, followed by heartbeat and respiratory arrest, electrocardiogram to restore sinus rhythm, but still no response, Need to use mechanical ventilation. During the next five days, urine output increased progressively to 7 liters / day. Serum sodium rose to 169 meq / l on day 8, serum osmolality was 339 osmolality / liter, and urine specific gravity was 1.005. Diabetes insipidus was diagnosed. The first 15 days with vasopressin 15 units of intramuscular injection, urine output decreased from 300 ml / hour to 85 ml / hour. 4 days after intermittent intramuscular injection is still maintained. Day 13 began to switch to intravenous infusion