临床上,阵发性心房颤动(房颤)随时间的推移,常演变为持久房颤,多数伴随左房的逐渐扩大。目前认为房颤时心房肌的电重构和随之产生的心房肌的纤维化是产生这一现象的主要原因,而血管紧张素Ⅱ介导的心房肌细胞内钙负荷过重可能是这一过程中的中心环节[1]。我们采用贝那普利和地尔 Clinically, paroxysmal atrial fibrillation (atrial fibrillation) over time, often evolve into a lasting atrial fibrillation, most with the gradual expansion of the left atrium. It is currently believed that the electrical remodeling of the atrial myocardium and consequent atrial fibrosis during atrial fibrillation are the major causes of this phenomenon, which may be the result of angiotensin II-mediated atrial calcium overload The central part of the process [1]. We use benazepril and dell