论文部分内容阅读
1例58岁男性患者因原发性高血压给予非洛地平缓释片5mg,1次/d;阿托伐他汀钙10mg,1次/d;维生素E0.1g,1次/d。3个月后血生化检查:K+3.11mmol/L。给予口服氯化钾缓释片,血钾恢复正常,但停用后又降低,遂入院。入院当日K+3.35mmol/L。经检查排除原发性醛固酮增多症所引起的低钾血症。停用非洛地平缓释片,改为厄贝沙坦0.15g,1次/d,并加用氯化钾缓释片0.5g,3次/d,其他治疗不变,2周后复查血生化示K+4.19mmol/L。停用氯化钾缓释片,1个月后K+3.85mmol/L,随后多次复查血钾均正常。
One 58-year-old man with essential hypertension was given felodipine sustained-release tablets 5 mg once daily, atorvastatin calcium 10 mg once daily and vitamin E 0.1 g once daily. Blood biochemistry examination after 3 months: K + 3.11 mmol / L. Given oral potassium chloride sustained release tablets, serum potassium returned to normal, but after the reduction and then reduced, then hospitalized. K + 3.35mmol / L on the day of admission. After examination excluded primary aldosteronism caused by hypokalemia. Stop using felodipine extended release tablets, to irbesartan 0.15g, 1 / d, and add potassium chloride sustained-release tablets 0.5g, 3 times / d, the other treatment unchanged, 2 weeks after the review of blood Biochemical showed K + 4.19mmol / L. Discontinuation of potassium chloride sustained release tablets, K + 3.85mmol / L after 1 month, followed by repeated review of serum potassium were normal.