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甲基汞除引起神经系统损伤外,有些学者还报道了动物接触甲基汞后,肾脏的近曲小管发生脂肪性变和坏死。Mag0s还指出慢性接触甲基汞的动物,肾脏中汞含量高于其他脏器。1977年Stroo等人发现慢性接触甲基汞的大鼠β-半乳糖酶和酸性磷酸酶的排泄量减少,但硷性磷酸酶未受影响。因此,在本文中,作者通过测定慢性接触甲基汞大鼠的肾脏和尿中硷性磷酸酶、乳酸脱氢酶、溶菌酶,谷氨酸脱氢酶和酸性磷酸酶的活性来解释甲基汞对肾脏损伤的机理。
In addition to methylmercury cause nervous system damage, some scholars have also reported that animal exposure to methylmercury, proximal tubule of the kidneys fatty degeneration and necrosis. Mag0s also pointed out that animals exposed to methylmercury on a chronic basis had higher levels of mercury in the kidneys than other organs. In 1977, Stroo et al. Found that the excretion of rat β-galactosidase and acid phosphatase in chronic exposure to methylmercury was reduced, but that of alkaline phosphatase was unaffected. Therefore, in this paper, the authors interpret the methyl groups by measuring the activity of alkaline phosphatase, lactate dehydrogenase, lysozyme, glutamate dehydrogenase and acid phosphatase in the kidneys and urine of rats exposed to methylmercury Mechanism of mercury on kidney damage.