8例先兆子痫胎盘绒毛的超微结构变化是合体滋养细胞退行性变,细胞滋养细胞增生。滋养层下的基底膜增厚,类纤维蛋白沉着及绒毛间质内的毛细血管肿胀,管腔变小等,以目前较流行的关于本病病因的子宫胎盘缺血及弥漫性血管内凝血学说可较合理地解释先兆子痫时胎盘超微结构所见为缺氧所致。 The ultrastructural changes of placental villi in 8 cases of preeclampsia were degenerative changes of syncytiotrophoblast cells and proliferation of cytotrophoblast cells. Under the trophoblast basement membrane thickening, fibrin-like protein deposition and villus capillary villi, lumen smaller, with the more popular on the etiology of the uterine placental ischemia and diffuse intravascular coagulation theory Placental ultrastructure can be more reasonable to explain the pre-eclampsia seen due to hypoxia.