近年来由于治疗学的进步,急性出血性胰腺炎死于休克和肾功能衰竭等早期并发症者有所减少;因而并发呼吸衰竭成为一个突出的问题,其死亡率在6.7～58%之间。本文就此作一简要概述。发病机理:很久以来一直认为急性胰腺炎中肺功能不全的原因是由于横膈抬高、胸膜渗液、支气管肺炎和肺动脉栓塞。然而,急性呼吸功能不全也能发生在无上述并发症的急性胰腺炎患者中,其可能解释有: 肺功能改变:1.肺换气不足,由于腹胀、腹痛和/或胸膜渗液。2.高呼出量呼吸衰竭,这类呼吸衰竭发生在腹膜炎和肠梗阻早期。尽管病初即出现呼吸加速,但氧饱和度仍不能满足组 In recent years due to advances in the treatment of acute hemorrhagic pancreatitis died of shock and renal failure and other early complications have decreased; thus complicated by respiratory failure has become a prominent problem, the mortality rate of 6.7% to 58%. This article gives a brief overview of this. Pathogenesis: It has long been assumed that the cause of pulmonary insufficiency in acute pancreatitis is due to diaphragmatic elevation, pleural effusion, bronchopneumonia, and pulmonary embolism. However, acute respiratory insufficiency can also occur in patients with acute pancreatitis without the above complications, which may explain: changes in lung function: 1. inadequate ventilation of the lung due to bloating, abdominal pain and / or pleural exudate. 2. Shout out respiratory failure, such respiratory failure occurs in the early peritonitis and intestinal obstruction. Oxygen saturation still did not meet the group despite rapid respiratory arrest at the beginning of the disease