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在对十种尘肺(典型矽肺、非典型矽肺、急性进展型矽肺、石棉肺、滑石肺、蜡石肺、硅藻土尘肺、金属铝肺、三氧化二铝肺、黄铁矿尘肺、黄铁矿熔渣尘肺、焊工尘肺、炭肺、活性炭肺、石墨肺及煤工尘肺)严谨地进行组织学研究的基础上,作者对粉尘灶的发生机理,作如下的推断: Ⅰ、石棉肺: 吸入之石棉长纤维刺激呼吸性细支气管或其它细支气管而发生细支气管炎。 细支气管的炎症伴随如下两个主要过程: 1)细支气管的炎症容易影响邻近的肺泡,产生纤维化及使呼吸性细支气管扩张,终于形
After ten types of pneumoconiosis (typical silicosis, atypical silicosis, acute progressive silicosis, asbestosis, talc, waxstone, diatomaceous earth pneumoconiosis, aluminum metal lung, aluminum oxide lung, pyrite pneumoconiosis, Based on the rigorous histological study, the author made the following conclusions on the mechanism of occurrence of dust foci: Ⅰ. Asbestos Pulmonary: Inhalation Asbestos long fibers stimulate respiratory bronchioles or other bronchioles and bronchiolitis occurs. Bronchial inflammation is accompanied by the following two main processes: 1) Bronchioal inflammation tends to affect adjacent alveoli, fibrosis and respiratory bronchioles, finally