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为研究心传导系统（CCS）与猝死的相互关系，对120例心性猝死者CCS作常规检查，光镜下作半定量计算其脂肪含量。结果发现：9例经一般常规尸检未找到任何致死原因的青年人（30岁以下），其房室结（AVN）与房室束（HB）的脂肪组织含量为70％，比同龄人明显增多，致使结、束肌细胞被脂肪分割中断或压迫萎缩。结内外细胞连接不足或缺失是构成心电不稳的病理基础。此外本文尚对其脂肪浸润的病因发病，猝死机理作了探讨。
In order to study the relationship between cardiac conduction system (CCS) and sudden death, CCS of 120 cases of sudden cardiac death were routinely examined, and the fat content was semi-quantitatively calculated under light microscope. The results showed that: Among 9 young adults (under 30 years old) who did not find any causes of death by routine autopsy, their AV contents and HBs were 70% higher than their peers , Resulting in knot, bundle muscle cells interrupted by fat partition or compression atrophy. Insufficient cell junction or lack of connectivity is the pathological basis for unstable ECG. In addition, this article still on its etiology of fat infiltration, sudden death mechanism is discussed.