细胞生物学领先杂志美国《细胞报告》刊登的一项新研究发现,细胞内铁过量与老年黄斑变性(AMD)之间存在关联。研究人员描述了视网膜细胞内铁聚集导致AMD的分子机制。新研究由美国肯塔基大学研究员布拉德利·盖尔范德博士及其同事合作完成。研究人员表示,铁是人体细胞维持正常代谢过程的不可或缺的微量元素。然而,最新研究发现,铁过量(“铁超负荷”)则会对细胞和组织构成严重伤害,而且会导致包括AMI)在内的多种疾病。尽管铁过量 A new study published in the Cell Report in the United States, the leading cell biology journal, found that there was a link between excessive intracellular iron and AMD. The researchers described the molecular mechanisms by which iron in the retina’s intracellular aggregates lead to AMD. The new study was done in collaboration with Dr. Bradley Gelvard, a researcher at the University of Kentucky and colleagues. Researchers say that iron is an essential trace element for the normal metabolism of human cells. However, the latest study found that iron overload (“iron overload”) can cause serious damage to cells and tissues and can lead to a variety of diseases including AMI. Despite iron overload