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目的探讨川崎病急性期血液粘度变化的意义。方法观察20例川崎病急性期患儿血液粘度的变化,并对红细胞聚集性、血小板聚集性、血浆纤维蛋白原、α2球蛋白等影响因素进行了分析。结果川崎病急性期全血低切粘度增高(P<0.01),红细胞聚集性、血小板聚集性增加(P<0.01,P<0.05),血浆粘度、血浆纤维蛋白原和α2球蛋白均增高(P<0.05,P<0.05,P<0.01)。全血低切粘度随红细胞聚集性增强而增高,与血浆粘度和血小板聚集性呈正相关(r=0.50,r=0.45,P均<0.05)。红细胞聚集性与血浆纤维蛋白原、α2球蛋白呈正相关(r=0.62,r=0.84,P均<0.01),血浆粘度与纤维蛋白原、α2球蛋白呈正相关(r=0.74r=0.56,P均<0.01)。结论川崎病急性期红细胞聚集性、血小板聚集性和血浆粘度增高导致全血低切粘度增加,对临床有指导意义。
Objective To explore the significance of blood viscosity changes in acute Kawasaki disease. Methods The changes of blood viscosity in 20 children with acute Kawasaki disease were observed and the influencing factors such as erythrocyte aggregation, platelet aggregation, plasma fibrinogen and α2 globulin were analyzed. Results In the acute stage of Kawasaki disease, the low blood viscosity of whole blood was increased (P <0.01), the aggregation of erythrocytes and platelet aggregation (P <0.01, P <0.05) and plasma viscosity, plasma fibrinogen and α2 globulin were increased <0.05, P <0.05, P <0.01). Low shear viscosity of whole blood increased with the increase of erythrocyte aggregation, and positively correlated with plasma viscosity and platelet aggregation (r = 0.50, r = 0.45, P <0.05). Erythrocyte aggregation was positively correlated with plasma fibrinogen and α2 globulin (r = 0.62, r = 0.84, P <0.01). Plasma viscosity was positively correlated with fibrinogen and α2 globulin (r = 0.74 r = 0.56, P All <0.01). Conclusion Kawasaki disease acute erythrocyte aggregation, platelet aggregation and increased plasma viscosity lead to increased viscosity of whole blood low cut, which is of clinical significance.