特发性高血压的人比正常血压的对照者很可能有类风湿因子、甲状腺抗体及其他自身免疫抗体,血清中的 IgG 及 IgA 浓度增高。一种似乎合理的解释是,这种免疫学的变化是由高血压或治疗高血压药物所造成的组织损害所引起。一种更为有趣的假说是,特发性高血压与某些免疫学的变化共同具有遗传的和环境的原因。这两种原因之一使得 People with idiopathic hypertension are more likely to have rheumatoid factor, thyroid antibodies and other autoimmune antibodies than their normotensive counterparts, with elevated serum IgG and IgA levels. One plausible explanation is that this immunological change is caused by tissue damage caused by high blood pressure or the treatment of high blood pressure medications. A more interesting hypothesis is that idiopathic hypertension and certain immunological changes have both genetic and environmental causes. One of these two reasons makes it