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线粒体是心肌保护的重要靶器官。随着对缺血再灌注损伤的机制研究的深入，线粒体ATP敏感性钾通道的开放和线粒体通透性转化孔的关闭被认为是治疗缺血再灌注损伤最有前途的靶位。“,”The focal point for myocardial protection is mitochondria .Studies on the prevention of ischemia-reperfusion injury involving the activation of mitochondrial ATP-sensitive potassium channels and the inhibition of mitochondrial permeability transition pore opening , have shown these to be promising therapeutic targets .