重症肌无力是神经肌肉接头处传递障碍的慢性疾病,也就是说支配肌肉收缩的神经在多种病因的影响下,不能将“信号”正常传递到肌肉,使肌肉丧失了收缩功能。通过现代医学的不断研究发现,重症肌无力的发病是自身免疫引起的。在正常情况下,运动神经末端合成乙酰胆碱,乙酰胆碱在肌肉收缩过程中起着决定性作用,它的含量减少或者受体数减少、敏感性降低均可以 Myasthenia gravis is a chronic disease at the neuromuscular junction that impairs the delivery of the disorder. That is to say, the nerves that dominate the muscle contraction can not transmit the “signal” to the muscle normally under the influence of various etiologies, resulting in loss of muscle contraction. Through the continuous study of modern medicine found that the incidence of myasthenia gravis caused by autoimmune. Under normal circumstances, synthesis of acetylcholine at the motor nerve end, acetylcholine play a decisive role in the process of muscle contraction, its reduced content or reduced number of receptors, the sensitivity can be reduced