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用结扎大鼠左冠状动脉前降支的方法造成心肌梗死病变模型，给予中药心复康后对其病理形态的改变进行观测，结果显示可使实验性心肌梗死大鼠梗死程度减轻，梗死后修复增快。采用体外培养的大鼠乳鼠心肌细胞，通过暂时停止供应心肌细胞生存必需的糖和氧，造成心肌细胞缺氧样损伤，从细胞水平观察心复康的作用，结果表明可明显改善心肌细胞结构缺血样损伤。提示心复康对心肌梗死具有防治作用。
The rat model of myocardial infarction was created by ligation of the left anterior descending coronary artery of the left ventricle. The pathological changes of the heart were observed after administration of Xinfu Kang. The results showed that the degree of infarction in experimental myocardial infarction rats could be reduced and repaired after infarction. Faster. Using rat cardiomyocytes cultured in vitro, by temporarily stopping the supply of cardiomyocytes for the survival of necessary sugars and oxygen, causing myocardial hypoxia-like injury, the effect of Xinfukang was observed from the cellular level. The results showed that the myocardial cell structure can be significantly improved. Ischemia-like damage. It is suggested that Xinfukang has preventive and therapeutic effects on myocardial infarction.