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随着药物、介入、外科治疗水平的提高,心肌梗死的病死率有所下降,但由于负性心室重构,很多心肌梗死存活患者出现进行性心功能下降。急性心肌梗死后数天内绝大部分心肌细胞呈凝固性坏死,随后大量炎性细胞浸润梗死区,溶解和吸收坏死组织,并大量分泌基质金属蛋白酶等活性物质。基
With drug, intervention, surgical treatment increased, the mortality rate of myocardial infarction decreased, but because of negative ventricular remodeling, many myocardial infarction patients with progressive cardiac decline. A large number of myocardial cells within a few days after acute myocardial infarction coagulation necrosis, followed by a large number of infiltration of inflammatory cells in the infarct zone, dissolved and absorbed necrotic tissue, and a large number of secretion of matrix metalloproteinase and other active substances. base