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激肽系统包括激肽以及与它的形成和分解有关的物质。激肽原在激肽释放酶的影响下释放激肽,而激肽酶可使后者失活。本文观察56例银屑病和41名健康人。测定血液中缓激肽原、游离激肽的含量,血浆激肽酶的活性,以及皮肤破坏激肽的能力。进行期血液中缓激肽原含量下降,游离缓激肽增多而血浆激肽酶活性无变化。静止期和退行期缓激肽原和游离缓激肽趋向正常。动态观察16例,其中11例在皮损消退时血浆激肽酶活性增高。缓激肽原是激肽的非活性前身,存在于血浆α_2球蛋白中。当肝功能正常时缓激肽原含量降低说明了对它的消耗和产生游离缓激肽的量增加。血
The kinin system includes kinins and substances related to its formation and breakdown. Kinin releases kinin under the action of kallikrein, which can inactivate the latter. This article observed 56 cases of psoriasis and 41 healthy people. Determination of bradykinin in blood, free kinin content, plasma kallikrein activity, and the ability of the skin to destroy kinin. During the period of blood bradykinin content decreased, increased free bradykinin plasma kallikinase activity without change. Rest and degenerative bradykinin and free bradykinin tended to be normal. Dynamic observation of 16 cases, of which 11 cases of skin lesions subsided plasma kallikinase activity increased. Bradykinin is an inactive precursor of kinins and is found in plasma alpha 2-globulin. A decrease in the content of bradykinin when liver function is normal indicates an increase in its consumption and production of free bradykinin. blood