DNA是重要的辐射靶分子,细胞受到照射后DNA大分子的链断裂,特别是双链断裂,被认为是细胞死亡的主要原因之一。我们的实验已经证明二乙基硫辛酰胺(“抗110”)对造血组织具有良好的而且是直接的保护作用,并且也已证明对受照骨髓细胞DNA的S期合成和非预定合成都有促进作用。因此,有理由设想“抗110”的抗放效价也可能在减少骨髓细胞DNA的链断裂或加速其重接修复上有所反映。我们已用国产 DNA is an important target of radiation. One of the main reasons for cell death is that the DNA is broken by DNA, especially double-strand breaks. Our experiments have demonstrated that diethyl-lipoamide (“anti-110”) has a good and direct protective effect on hematopoietic tissues and has also demonstrated that both S-phase synthesis and non-scheduled synthesis of DNA from irradiated myeloid cells have enhancement. Therefore, it is reasonable to assume that the anti-putative potency of “anti-110” may also be reflected in the reduction of DNA strand breaks or the accelerated reconnection of bone marrow cells. We have used domestic