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本文简要地概括了肝昏迷机制研究的现状。较详细地论述和评价了氨代谢障碍与肝昏迷的关系。概述了近10年来提出的几个新机制——低级脂肪酸、假神经介质、血浆氨基酸不平衡与肝昏迷的关系。分析了诱发因素和临床治疗的理论根据。
This article briefly summarizes the current status of hepatic coma. A more detailed discussion and evaluation of the relationship between ammonia metabolism disorders and hepatic coma. Several new mechanisms proposed in recent ten years are summarized: low-grade fatty acid, pseudo-neural medium, plasma amino acid imbalance and hepatic coma. The theoretical basis of predisposing factors and clinical treatment were analyzed.