论文部分内容阅读
幽门螺杆菌(HP)感染已被公认为是慢性胃炎的主要病因.目前,人们讨论的中心问题是其致病机制,究竟是细菌直接侵袭胃粘膜上皮细胞诱发局部组织损伤,或是细菌产生的毒素作用于局部或远隔区域,或是细菌代谢产物及细菌本身引起的免疫病理反应等尚无定论.本文通过对167例慢性胃炎患者胃窦部粘膜组织快速尿素酶试验及石蜡包埋病理切片,对照观察HP感染量与慢性胃炎程度之间的关系.以期从量的方面了解HP感染与慢性胃炎的关系,进一步加深对慢性胃炎发生机制的理解.
Helicobacter pylori (HP) infection has been recognized as the main cause of chronic gastritis.Currently, the central issue people are discussing is its pathogenic mechanism, whether bacterial direct invasion of gastric mucosal epithelial cells induced by local tissue damage, or bacteria Toxin in local or remote areas, or bacterial metabolites and the bacteria itself caused by the immune pathological response is inconclusive.In this paper, 167 cases of chronic gastritis mucosa in patients with rapid urease test and paraffin-embedded sections , To observe the relationship between the HP infection and the degree of chronic gastritis.Objective To understand the relationship between HP infection and chronic gastritis in terms of quantity, and to further deepen the understanding of the mechanism of chronic gastritis.