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[会议论文] 作者：Yangli Xie,Nan Su,Min Jin,Can Li,Lin chen,
来源：第二届模式生物与人类健康研讨会 年份：2012
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[会议论文] 作者：Yangli Xie,Nan Su,Min Jin,Can Li,Lin chen,
来源：第二届模式生物与人类健康研讨会 年份：2012
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[会议论文] 作者：Lin Chen,Min Jin,Yangli Xie,NanSu,Huabing Qi,XiaoFeng Wang,
来源：第六届国际骨质疏松及骨矿盐疾病学术会议 年份：2012
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[会议论文] 作者：Yangli Xie,Nan Su,Siru Zhou,Huabing Qi,Junlan Huang,Lin Chen,
来源：2012全国发育生物学大会 年份：2012
Fracture healing is a postnatal repair process which recapitulates of skeletal development.FGFR3 is a negative regulator of chondrogenesis during embryo sta...
[会议论文] 作者：Yangli Xie,Siru Zhou,Nan Su,Min Jin,Can Li,Lin Chen,
来源：第六届国际骨质疏松及骨矿盐疾病学术会议 年份：2012
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[会议论文] 作者：Jie Yin,Lele Yang,Yangli Xie,Hongbin Ji,Lin Chen,Ping Hu,
来源：中国生物化学与分子生物学会第十二届会员代表大会暨2018年全国学术会议 年份：2018
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Exogenous PTH1-34 can rescue the delayed fracture healing caused by gain-of function mutation of FGF
[会议论文] 作者：yangli xie,siru zhou,wei xu,zuqiang wang,junlan huang,xiaolan du,lin chen,
来源：中华医学会第八次全国骨质疏松和骨矿盐疾病学术会议 年份：2015
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Exogenous PTH1-34 can rescue the delayed fracture healing caused by gain-of-function mutation of FGF
[会议论文] 作者：Yangli Xie,Nan Su,Siru Zhou,Min Jin,Huabing Qi,Can Li,Lin Chen,
来源：2012全国发育生物学大会 年份：2012
Achondroplasia(ACH) is the most common form of dwarfism caused by activated mutations of FGFR3 gene.Gain-of-function mutation of FGFR3 in mice (FGFR3G369C/+...
Exogenous PTH1-34 can rescue the delayed fracture healing caused by gain-of function mutation of FGF
[会议论文] 作者：yangli xie,siru zhou,wei xu,zuqiang wang,junlan huang,xiaolan du,lin chen,
来源：中华医学会第八次全国骨质疏松和骨矿盐疾病学术会议 年份：2015
Achondroplasia (ACH) is the most common form of dwarfism caused by activated mutations of FGFR3 gene.Gain-of-function mutation of FGFR3 in mice (FGFR3G369C/+ mice,ACH mice) resulted in delayed fractur...
Spatially defined single-cell transcriptional profiling characterizes diverse chondrocyte subtypes a
[期刊论文] 作者：Yibo Gan,Jian He,Jun Zhu,Zhengyang Xu,Zhong Wang,Jing Yan,Ou Hu,Zhijie Bai,Lin Chen,Yangli Xie,Min Jin,
来源：骨研究:英文版 年份：2021
A comprehensive understanding of the cellular heterogeneity and molecular mechanisms underlying the development,homeostasis,and disease of human intervertebral disks(IVDs)remains challenging.Here,the transcriptomic landscape of 108108 IVD c......
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