继发于感染和炎症的贫血相当多见,由于注意力集中于原发疾病,往往被临床医生忽视。现就感染性贫血的有关问题作一介绍。病因:金黄色葡萄球菌、链球菌、流感杆菌、产气(竹夹)膜杆菌可引起溶血。疟原虫寄生于红细胞内,破坏红细胞亦产生溶血。慢性感染引起的贫血多见于结核病、伤寒、脓胸、肺脓肿,亚急性细菌性心内膜炎,慢性骨髓炎、肝脓肿以及其他化脓性疾病。胶原性疾病如类风湿性关节炎等伴发的贫血也归入这类贫血。病毒性肝炎常引起一时性血细胞生成异常,表现为白细胞、血小板减少或全血细胞减少,红细胞寿命缩短和溶血性贫血。随着肝炎好转而恢复正常。但有少数患者可发生再生障碍性贫血,国内已屡有报道。多数发生于急性黄疸型肝炎的恢复期。并发再障的肝炎约占同期肝炎的0.34—0.4％。再障发生时间多数在 Anemia secondary to infection and inflammation is quite common and is often overlooked by clinicians due to the concentration of the primary disease. Now on the issue of infectious anemia to make an introduction. Etiology: Staphylococcus aureus, Streptococcus, influenza bacilli, gas (bamboo clip) Bacillus can cause hemolysis. Plasmodium parasites in the red blood cells, destruction of red blood cells also produce hemolysis. Chronic infection-related anemia more common in tuberculosis, typhoid, empyema, lung abscess, subacute bacterial endocarditis, chronic osteomyelitis, liver abscess and other suppurative diseases. Collagenous diseases such as rheumatoid arthritis and other associated anemia also fall into this category of anemia. Often caused by viral hepatitis temporary hematopoietic anomalies, manifested as leukopenia, thrombocytopenia or pancytopenia, shortened life span of red blood cells and hemolytic anemia. With hepatitis turn back to normal. However, a small number of patients with aplastic anemia can occur, China has repeatedly reported. Most occurred in the recovery of acute jaundice hepatitis. Secondary aplastic anemia accounted for about 0.34-0.4% of hepatitis. Aplastic anemia occurs most of the time