家犬于石棉车间自然吸入染尘1～3年后,其呼吸性细支气管、肺泡管及膜性细支气管均显示不同程度的粉尘沉着伴纤维化。病变程度亦随染尘时间延长而逐渐加重,酷似人体石棉肺小气道病变。肺病灶内尘粒能谱分析揭示其病因相当复杂,原则上是一种混合粉尘,以硅酸盐为主,石棉占绝对优势,且闪石类多于温石棉,并存在明显剂量效应关系。 Domestic dogs in asbestos workshop natural inhalation of dust 1 to 3 years after the respiratory bronchioles, alveolar and membranous bronchioles showed varying degrees of dust deposition with fibrosis. The degree of lesion also gradually aggravate with the extension of the dust time, resembles the human asbestos pulmonary small airway disease. Spectra analysis of the dust particles in the lung lesion reveals that the etiology is quite complicated. In principle, it is a kind of mixed dust, silicate, asbestos, and more amphiboles than chrysotile, with obvious dose-effect relationship.