论文部分内容阅读
例1,男,100d,因发作性抽搐20d住当地医院。患儿既往身体健康,病程中无明显发热,抽搐间隙一般情况尚好。检查:脑脊液及脑电图正常。经大量补钙及止痉药物处理,症状未见好转,于住院第6d转我院。入院时体检,头颅五官无畸形,发育一般,营养欠佳,精神萎糜。前囟1×1 cm~2,平软。两肺闻及细湿罗音。心率108次,律齐,无杂音,心音低钝。肝肋下3.5cm,剑下4.5cm,质软。神经系统无异常。胸片提示心脏横径增大,两肺野斑片状模糊阴影,未见胸腺阴影。EKG示T波变化,左室高电压。WBC22.4×10~9/L,N0.60,L0.40。拟诊支气管肺炎、心源性脑缺氧综合征、维生素B_1缺乏症。给予抗感染、补钙等对症处理,抽搐始终未完全控制,后因肺部感染
Example 1, male, 100d, due to seizures twitch 20d live in a local hospital. In the past, children with good health, no fever in the course of the disease, twitch gap is generally good. Check: Cerebrospinal fluid and EEG normal. After a large number of calcium and antispasmodic drugs, the symptoms did not improve, in the hospital 6d transfer to our hospital. Physical examination on admission, head and facial features deformities, general development, poor nutrition, spiritual wilt Mi. Anterior fontanel 1 × 1 cm ~ 2, soft. Smell both lungs and wet rales. Heart rate 108 times, law Qi, no noise, low heart sound blunt. Liver ribs 3.5cm, sword under 4.5cm, soft. Nervous system no abnormalities. Chest x-ray tips increased diameter of the heart, two lungs patchy fuzzy shadow, no thymus shadow. EKG showed T wave changes, left ventricular high voltage. WBC22.4 × 10 ~ 9 / L, N0.60, L0.40. Proposed diagnosis of bronchial pneumonia, cardiogenic cerebral hypoxemia syndrome, vitamin B_1 deficiency. Given anti-infective, calcium and other symptomatic treatment, twitch has not been fully controlled, because of lung infection