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川崎病是一种病因不明的以全身血管炎症为病理改变的自限性疾病,以中、小血管病变为著,特别是冠状动脉。婴幼儿发病率高。1998年1月～2001年12月我们观察了川崎病患儿血清可溶性细胞间黏附分子-1(sICAM-1)的变化,以探讨细胞间黏附分子-1(ICAM-1)在本病发病机制中的作用。
Kawasaki disease is an unknown etiology of systemic vascular inflammation as a pathological change of self-limiting disease, with small and medium vascular lesions, especially the coronary arteries. High incidence of infants and young children. From January 1998 to December 2001, we observed the changes of sICAM-1 in children with Kawasaki disease to explore the role of ICAM-1 in the pathogenesis of this disease In the role.