尾状核出血常由于高血压,动-静脉畸形(AVM)或动脉瘤破裂引起,罕见于 moyamoya 病。严重的动脉硬化性闭塞性疾病所致的 moyamoya样的改变通常引起缺血性卒中。本文报道1例男性,51岁,白人,无高血压病史。有冠心病、高胆固醇血症、间歇性跛行及吸烟史。因突然剧烈头痛、恶心、呕吐一天入院,无神经系统局灶症状,体检除颈抵抗外余神经系正常。次日头颅 CT 示“左尾状核出血,破入脑室”。血管造影 Caudate nucleus haemorrhage often due to hypertension, arteriovenous malformation (AVM) or aneurysm rupture, rare in moyamoya disease. A moyamoya-like change caused by a serious atherosclerotic occlusive disease usually causes ischemic stroke. This article reports a male, 51 years old, white, no history of hypertension. Coronary heart disease, hypercholesterolemia, intermittent claudication and smoking history. Due to sudden severe headache, nausea, vomiting one day admission, no focal neurological symptoms, physical examination in addition to the normal resistance to the external nervous system. The next day head CT showed “left caudate nucleus bleeding, broken into the ventricle.” Angiography