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为阐明鼻呼吸在支气管肺系炎症过程发病机制中的作用,作者们用16只家兔进行了实验研究。经电灼下甲前端、中隔粘膜和鼻孔周围皮肤,但不缝合前鼻孔,5～6天后造成单侧鼻呼吸阻断。4个月后,全部实验动物放血致死。从气管、双侧主支气管和双侧肺上,下叶分取组织小块,行HE和苦复红染色。以淀粉酶控制的Schick氏反应确定所含中性粘多糖和糖原,以Hale-Steedman氏法显示酸性粘多糖。
To clarify the role of nasal breathing in the pathogenesis of bronchopulmonary inflammatory processes, the authors conducted an experimental study with 16 rabbits. Under the electrocautery of the A front, septal mucosa and the skin around the nostrils, but not before suturing the nose, 5 to 6 days after unilateral nasal respiratory block. Four months later, all experimental animals were lethal. Tissues from the trachea, bilateral main bronchus and bilateral upper and lower leaves were dissected for HE and Kufuhong staining. The neutral mucopolysaccharides and glycogen contained were determined by amylase-controlled Schick’s reaction and acid mucopolysaccharide was visualized by the Hale-Steedman’s method.