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β-葡萄糖醛酸甙酶(GRS)为细胞溶酶体中的酸性水解酶,正常情况下GRS主要由肾小管与尿路上皮分泌,或随细胞脱落进入尿液。膀胱肿瘤与其它部位尿路上皮癌其细胞溶酶体中GRS合成增加,恶性细胞崩解或细胞膜通透性改变,引起尿 GRS活性上升。本文对26例来自肾小管细胞恶变的肾腺癌患者尿GRS活性进行了观察,并与膀胱癌进行了比较,现报道如下。 对象与方法 一、对象:1982年~1984年肾肿瘤患者26例(男19,女7),年龄在50岁以上占81%。其中肾腺癌、肾间叶组织来源肿瘤、肾囊肿分别为18、4、4例。
Β-glucuronosidase (GRS) is an acidic hydrolase in the lysosome of cells. Under normal circumstances, GRS is mainly secreted from the renal tubules and the urothelium, or falls off into the urine as the cells. GRS synthesis in lysosomes of bladder cancer and other sites of urothelial carcinoma increases, malignant cells disintegrate or cell membrane permeability changes, causing urinary GRS activity to rise. In this paper, urinary GRS activity in 26 cases of renal adenocarcinoma patients with malignant changes of renal tubular cells was observed and compared with bladder cancer. The following are reported. Subjects and Methods 1. Object: 26 patients (19 males and 7 females) with kidney cancer from 1982 to 1984, aged 81 and above 81 accounted for 81%. Among them, renal adenocarcinoma, kidney-derived tissue tumors, and renal cysts were 18, 4, and 4 cases, respectively.