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据报道,SARS患者肺部的致病性依赖于疾病延续时间的长短。巨噬细胞和噬红细胞作用的存在提示,炎前细胞因子诱导过多的状态可能是对类固醇治疗的反应。 香港大学Peiris博士检查了6例SARS患者的尸检组织标本。由于这些患者死亡前病程至少持续了10天,肺组织已形成了毛玻璃膜、肺细胞增生和水肿。病程最长的患者其肺泡损坏形成病灶并扩散。
It has been reported that the pathogenicity of lungs in SARS patients depends on the duration of the disease. The existence of the role of macrophages and erythrophages suggests that pre-inflammatory cytokine-induced excess states may be a response to steroid treatment. Dr. Peiris from the University of Hong Kong examined autopsy tissue samples from 6 SARS patients. Since these patients last at least 10 days before their death, the lung tissue has formed a frosted glass membrane, lung cell proliferation and edema. The longest course of the patient’s alveoli damage the formation of lesions and spread.